Research Project:
Identification of TRAIL sensivity/resistance mechanisms and searching for novel TRAIL-sensitizing agents in Glioblastoma Multiforme

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dc.contributor.departmentGraduate School of Health Sciences
dc.contributor.departmentGraduate School of Health Sciences
dc.contributor.schoolcollegeinstituteGRADUATE SCHOOL OF HEALTH SCIENCES
dc.contributor.schoolcollegeinstituteGRADUATE SCHOOL OF HEALTH SCIENCES
dc.date.accessioned2024-12-29T10:37:44Z
dc.date.available2024-12-29
dc.description.publisherscopeInternational
dc.identifierEC.00058
dc.identifier.urihttps://hdl.handle.net/20.500.14288/24115
dc.relation.affiliationKoç University
dc.relation.collectionKoç University Projects Collection
dc.relation.relatedpublicationsThe pro-apoptotic Bcl-2 family member Harakiri (HRK) induces cell death in glioblastoma multiforme
dc.relation.relatedpublicationsKdm2b, an h3k36-specific demethylase, regulates apoptotic response of gbm cells to trail
dc.relation.relatedpublicationsThe fungal metabolite chaetocin is a sensitizer for pro-apoptotic therapies in glioblastoma
dc.relation.relatedpublicationsGeneration of TRAIL-resistant cell line models reveals distinct adaptive mechanisms for acquired resistance and re-sensitization
dc.titleIdentification of TRAIL sensivity/resistance mechanisms and searching for novel TRAIL-sensitizing agents in Glioblastoma Multiforme
dc.title.alternativeTRIGBM
dspace.entity.typeProject
local.contributor.kuauthorÖnder, Tuğba Bağcı
local.project.contractnumber618673
local.project.endDate2017-08-29
local.project.startDate2013-09-01
local.project.templateEC.FP07.PEOPLE.MCCIG
project.funder.identifierEC - FP7
project.funder.nameEuropean Commission
project.investigatorÖnder, Tuğba Bağcı
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