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Publication:
Endometriosis-derived iPSCs reveal conserved stromal maturation and endocrine responsiveness

dc.contributor.coauthorMcDowell, H.
dc.contributor.coauthorSun, S.
dc.contributor.coauthorMcNally, R.
dc.contributor.coauthorHuerta, C.
dc.contributor.coauthorAsif, H.
dc.contributor.coauthorYoon, J.
dc.contributor.coauthorAlvarez, A. A.
dc.contributor.coauthorFoley, K. G.
dc.contributor.coauthorBoots, C. E.
dc.contributor.coauthorMilad, M. P.
dc.contributor.coauthorKim, J.
dc.contributor.departmentSchool of Medicine
dc.contributor.kuauthorYıldız, Şule
dc.contributor.schoolcollegeinstituteSCHOOL OF MEDICINE
dc.date.accessioned2026-09-15T10:56:41Z
dc.date.issued2026
dc.description.abstractEndometriosis is a chronic, hormone-dependent disease characterized by altered endometrial stromal function, but mechanistic and translational studies have been hindered by the lack of tractable human models. Here, we establish an induced pluripotent stem cell (iPSC)–based platform derived from patients with endometriosis to model endometrial stromal differentiation in a controlled human context. Using a defined differentiation protocol, endometriosis-derived iPSCs transition from pluripotency through mesenchymal commitment toward stromal-like states and acquire transcriptional hormone responsiveness. Transcriptomic analyses reveal coordinated repression of pluripotency and proliferative programs with induction of stromal lineage signatures. Comparison with independent transcriptomic datasets demonstrated that in vitro–derived stromal cells progressively acquired gene expression profiles resembling eutopic endometrial stromal programs in endometriosis. Conditioned media from iPSC-derived stromal cells also induced transcriptional reprogramming in THP-1 macrophage-like cells. Together, these findings establish a patient-derived platform for investigating stromal differentiation and stromal-immune interactions in endometriosis.
dc.description.harvestedfromManual
dc.description.indexedbyPubMed
dc.description.indexedbyScopus
dc.description.publisherscopeInternational
dc.description.sponsoredbyTubitakEuN/A
dc.description.sponsorshipNIH/NCI (Grant: CA009560); NIH/NIEHS (Grant: UH3 ES029073); NIH/NICHD (Grant: R01HD114195); Endometriosis Foundation; Friends of Prentice Foundation [Funding]: We are grateful to the following funding agencies: Friends of Prentice (J.J.K.), Endometriosis Foundation (J.J.K.), Eunice Kennedy Shriver National institute of Child Health and Human Development grant R01HD114195 (J.J.K.), National Institute of Environmental Health Sciences grant UH3 ES029073 (J.J.K.), and NCI NIH T32 training grant CA009560 (K.G.F.). [Acknowledgements]: We would like to acknowledge the Northwestern University NUSeq Core for sequencing and the Northwestern University Stem Cell Core for reprogramming the PBMCs to iPSCs. Funding: We are grateful to the following funding agencies: Friends of Prentice (J.J.K.), Endometriosis Foundation (J.J.K.), Eunice Kennedy Shriver National institute of Child Health and Human Development grant R01HD114195 (J.J.K.), National Institute of Environmental Health Sciences grant UH3 ES029073 (J.J.K.), and NCI NIH T32 training grant CA009560 (K.G.F.). Author contributions: Conceptualization: H.M., M.M., and J.J.K. Methodology: H.M., S.Y., R.M., S.S., A.A., C.H., J.Y., K.G.F., and J.J.K. Investigation: HD, C.B., H.A., S.Y., R.M., S.S., A.A., C.H., J.Y., K.G.F., and J.J.K. Visualization: HD, C.H., and J.J.K. Supervision: M.M. and J.J.K. Writing—original draft: HD and J.J.K. Writing—review and editing: HD, C.B., H.A., S.Y., R.M., S.S., A.A., C.H., J.Y., K.G.F., and J.J.K. Competing interests: The authors declare that they have no competing interests. Data, code, and materials availability: All data and code needed to evaluate and reproduce the results in the paper are present in the paper and/or the Supplementary Materials. Sequencing data were submitted to GEO ( GSE328997 ). The iPSC lines generated and used in this study can be provided by the corresponding author pending scientific review and a completed material transfer agreement. Requests for the iPSC lines should be submitted to the corresponding author J.J.K. (j-kim4@northwestern.edu).
dc.description.versionPublished Version
dc.identifier.ScopusPercentile96
dc.identifier.ScopusQuartileQ1
dc.identifier.WoSPercentile91.8
dc.identifier.WoSQuartileQ1
dc.identifier.doi10.1126/sciadv.aeg2362
dc.identifier.endpage-
dc.identifier.grantnoCA009560
dc.identifier.grantnoUH3 ES029073
dc.identifier.grantnoR01HD114195
dc.identifier.issn2375-2548
dc.identifier.issue34
dc.identifier.pubmed42616893
dc.identifier.scopus2-s2.0-105047932049
dc.identifier.startpage-
dc.identifier.urihttp://doi.org/10.1126/sciadv.aeg2362
dc.identifier.urihttps://hdl.handle.net/20.500.14288/35514
dc.identifier.volume12
dc.keywordsStromal cell
dc.keywordsReprogramming
dc.keywordsInduced pluripotent stem cell
dc.keywordsTranscriptome
dc.keywordsMesenchymal stem cell
dc.keywordsCellular differentiation
dc.keywordsStem cell
dc.keywordsLineage (genetic)
dc.languageeng
dc.publisherAmerican Association for the Advancement of Science (AAAS)
dc.relation.affiliationKoç University
dc.relation.collectionKoç University Institutional Repository
dc.relation.ispartofScience Advances
dc.relation.openaccessN/A
dc.subjectHealth sciences
dc.subjectMedicine
dc.subjectReproductive medicine
dc.subjectLife sciences
dc.subjectImmunology and microbiology
dc.subjectImmunology
dc.subjectObstetrics and gynecology
dc.titleEndometriosis-derived iPSCs reveal conserved stromal maturation and endocrine responsiveness
dc.typeJournal Article
dspace.entity.typePublication
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relation.isOrgUnitOfPublication.latestForDiscoveryd02929e1-2a70-44f0-ae17-7819f587bedd
relation.isParentOrgUnitOfPublication17f2dc8e-6e54-4fa8-b5e0-d6415123a93e
relation.isParentOrgUnitOfPublication.latestForDiscovery17f2dc8e-6e54-4fa8-b5e0-d6415123a93e

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