Publication:
Renin-angiotensin system and cancer: epidemiology, cell signaling, genetics and epigenetics

dc.contributor.coauthorAfsar, B.
dc.contributor.coauthorAfsar, R. E.
dc.contributor.coauthorKuwabara, M.
dc.contributor.coauthorOrtiz, A.
dc.contributor.coauthorCovic, A.
dc.contributor.departmentSchool of Medicine
dc.contributor.kuauthorErtuğlu, Lale Aslıhan
dc.contributor.kuauthorKanbay, Mehmet
dc.contributor.schoolcollegeinstituteSCHOOL OF MEDICINE
dc.date.accessioned2024-11-09T23:29:02Z
dc.date.issued2021
dc.description.abstractDay by day, the health and economical burden of cancer increases globally. Indeed it can be considered that there is ''cancer pandemic''. Blocking the renin-angiotensin system (RAS) by angiotensin-converting enzyme (ACE) inhibitors (ACEI) or angiotensin-receptor blockers (ARB) are widely used measures to treat hypertension and heart failure. It has been recently suggested the activation and blocking of RAS has been associated with various types of cancer in epidemiological and experimental studies. Various studies have shown that RAS blockage is protective in some cancers. However, although fewer, contradictory data also showed that RAS blockage is either not related or adversely related to cancer. Although the reasons for these findings are not exactly known, different types of receptors and effectors in RAS may account for these findings. In the current review, we summarize the different RAS receptors and cancer development with regard to epidemiology, and pathogenesis including cell signaling pathways, apoptosis, genetic and epigenetic factors.
dc.description.indexedbyWOS
dc.description.indexedbyScopus
dc.description.indexedbyPubMed
dc.description.issue4
dc.description.openaccessNO
dc.description.publisherscopeInternational
dc.description.sponsoredbyTubitakEuN/A
dc.description.sponsorshipPresidency of Turkey, Presidency of Strategy and Budget MK gratefully acknowledge the use of the services and facilities of the Koc University Research Center for Translational Medicine (KUTTAM), funded by the Presidency of Turkey, Presidency of Strategy and Budget. The content is solely the responsibility of the authors and does not necessarily represent the official views of the Presidency of Strategy and Budget.
dc.description.volume23
dc.identifier.doi10.1007/s12094-020-02488-3
dc.identifier.eissn1699-3055
dc.identifier.issn1699-048X
dc.identifier.quartileQ3
dc.identifier.scopus2-s2.0-85090928774
dc.identifier.urihttps://doi.org/10.1007/s12094-020-02488-3
dc.identifier.urihttps://hdl.handle.net/20.500.14288/11974
dc.identifier.wos569555700001
dc.keywordsAngiotensin
dc.keywordsApoptosis
dc.keywordsCancer
dc.keywordsCell signaling
dc.keywordsGenetic
dc.keywordsConverting enzyme-inhibitors
dc.keywordsII receptor blocker
dc.keywordsEndothelial growth-factor
dc.keywordsHuman pancreatic-cancer
dc.keywordsPopulation-based cohort
dc.keywordsBreast-cancer
dc.keywordsInsertion/deletion polymorphism
dc.keywordsColorectal-cancer
dc.keywordsAntiproliferative activity
dc.keywordsTyrosine kinase
dc.language.isoeng
dc.publisherSpringer
dc.relation.ispartofClinical and Translational Oncology
dc.subjectOncology
dc.titleRenin-angiotensin system and cancer: epidemiology, cell signaling, genetics and epigenetics
dc.typeReview
dspace.entity.typePublication
local.contributor.kuauthorKanbay, Mehmet
local.contributor.kuauthorErtuğlu, Lale Aslıhan
local.publication.orgunit1SCHOOL OF MEDICINE
local.publication.orgunit2School of Medicine
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