Publication:
Phenotypical spectrum of SACS variants: Neuromuscular perspective of a complex neurodegenerative disorder

dc.contributor.coauthorÇakar, Arman
dc.contributor.coauthorİnci, Meltem
dc.contributor.coauthorAcarlı, Ayşe Nur Özdağ
dc.contributor.coauthorComu, Sinan
dc.contributor.coauthorCandayan, Ayşe
dc.contributor.coauthorBattaloğlu, Esra
dc.contributor.coauthorBaşak, Durmuş, Hacer
dc.contributor.coauthorParman, Yeşim
dc.contributor.departmentKUTTAM (Koç University Research Center for Translational Medicine)
dc.contributor.departmentSchool of Medicine
dc.contributor.departmentNDAL (Neurodegeneration Research Laboratory)
dc.contributor.facultymemberYes
dc.contributor.kuauthorBaşak, Ayşe Nazlı
dc.contributor.kuauthorTekgül, Şeyma
dc.contributor.schoolcollegeinstituteResearch Center
dc.contributor.schoolcollegeinstituteSCHOOL OF MEDICINE
dc.contributor.schoolcollegeinstituteLaboratory
dc.date.accessioned2024-11-09T23:39:53Z
dc.date.issued2022
dc.description.abstractObjectives Autosomal recessive spastic ataxia of Charlevoix-Saguenay (ARSACS) is caused by the SACS gene variants. Main clinical features include early-onset and progressive cerebellar ataxia, spasticity, sensorimotor polyneuropathy. However, the phenotypic spectrum expanded with the increased availability of next-generation sequencing methods. Materials and Methods Herein, we describe the clinical features of nine patients from seven unrelated families with SACS variants from the cohort of the Neuromuscular Disorders Unit of the Neurology Department of the Istanbul University, Istanbul Faculty of Medicine. Results Seven patients were male. Seven patients in our cohort had disease onset in the first decade of life. Eight patients were born to consanguineous marriages. Distal weakness in the lower limbs was a prominent feature in all of our patients. Seven patients had ataxia, and six patients had spasticity. Interestingly, one patient showed an isolated Charcot-Marie-Tooth-like phenotype. Five patients showed sensorimotor demyelinating polyneuropathy in the nerve conduction studies. Linear pontine hypointensity was the most frequent cranial magnetic resonance imaging (MRI) abnormality. Two patients with a later disease onset had a homozygous c.11542_11544delATT (p.Ile3848del) variant. The rest of the identified variants were scattered throughout the SACS gene. Conclusions Atypical clinical features in our patients highlight that the phenotypic spectrum of ARSACS can be observed in a wide range.
dc.description.fulltextNo
dc.description.harvestedfromManual
dc.description.indexedbyWOS
dc.description.indexedbyScopus
dc.description.indexedbyPubMed
dc.description.openaccessNO
dc.description.peerreviewstatusN/A
dc.description.publisherscopeInternational
dc.description.readpublishN/A
dc.description.sponsoredbyTubitakEuN/A
dc.description.studentonlypublicationNo
dc.description.studentpublicationNo
dc.description.versionN/A
dc.identifier.doi10.1111/ane.13592
dc.identifier.eissn1600-0404
dc.identifier.embargoN/A
dc.identifier.endpage626
dc.identifier.issn0001-6314
dc.identifier.issue5
dc.identifier.pubmed35130357
dc.identifier.quartileQ3
dc.identifier.scopus2-s2.0-85124518608
dc.identifier.startpage619
dc.identifier.urihttps://doi.org/10.1111/ane.13592
dc.identifier.urihttps://hdl.handle.net/20.500.14288/13183
dc.identifier.volume145
dc.identifier.wos000751949200001
dc.keywordsARSACS
dc.keywordsAtaxia
dc.keywordsGenetic
dc.keywordsNeuropathy
dc.keywordsPolyneuropathy
dc.keywordsSpasticity recessive spastic ataxia
dc.keywordsMutations
dc.keywordsArsacs
dc.keywordsGene
dc.language.isoeng
dc.publisherWiley
dc.relation.affiliationKoç University
dc.relation.collectionKoç University Institutional Repository
dc.relation.ispartofActa neurologica scandinavica
dc.relation.openaccessN/A
dc.rightsN/A
dc.subjectClinical Neurology
dc.titlePhenotypical spectrum of SACS variants: Neuromuscular perspective of a complex neurodegenerative disorder
dc.typeJournal Article
dspace.entity.typePublication
local.contributor.kuauthorTekgül, Şeyma
local.contributor.kuauthorBaşak, Ayşe Nazlı
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